148 Bilder zum Thema "electrocardiogram education" bei ClipDealer

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gradient ecg
Bidirectional ventricular tachycardia is a kind of malignant arrhythmia. The polarity of QRS main wave alternates from beat to beat, and it is easy to degenerate into ventricular fibrillation.
The illustration shows the two patterns of ventricular tachycardia episodes.The green circle represents sinus rhythm. Picture A shows paroxysmal episodes of ventricular tachycardia, and picture B shows short bursts.
The left main coronary artery can be divided into the left anterior descending artery and the left circumflex artery, and sometimes the intermediate branch artery.
Due to the large mass of the left ventricle, the dominant excitation potentials of the left and right ventricles are oriented towards the left ventricle, i.e. towards the left, Inferior and posterior.
When there is a the first degree interatrial block, the impulse from the right atrium is slowly transmitted to the left atrium, causing widening notched P wave.
The transverse vectorcardiogram generates a chest leads electrocardiogram, with the maximum ventricular excitation potential oriented towards the left posterior region.
When the rhythm of the atria originates in the lower part of the atria, the whole atria are excited from inferior to superior, producing negative P waves in the inferior leads.
The terminal excitation of the ventricle forms the final part of the S wave in lead V1, gradually returning to the isoelectric line, and forms a small S wave in lead V5.
Under normal circumstances, in the chest lead electrocardiogram, the amplitude of the R wave gradually increases from lead V1 to lead V6.
When the sinus P wave on the ecg disappears, it may be due to abnormal generation and/or conduction of sinus impulses, or it may be due to atrial muscle lesions that cannot excite.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract forms a high amplitude R wave in the inferior leads and a QS wave or rS wave in the V1 lead.
When the first degree interatrial block occurs, the conduction time from the right atrium to the left atrium is prolonged, the P wave widens, and bimodal P wave ECG changes appear.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
The initial excitation of the ventricle forms a small r wave in lead V1 and a small q wave in lead V6.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
Ventricular tachycardia originating from the right ventricular outflow tract can be sustained or short-burst, and is a benign idiopathic ventricular tachycardia.
During normal ventricular excitation, the earliest epicardial breakthrough point is located in the paraventricular septal area, and the RV outflow tract and the base of the LV are finally excited.
A 2:1 left bundle branch block is considered when complete left bundle branch block alternates with normal QRS complexes and the PR interval is fixed.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
The conduction in ventricle is mainly divided into right bundle branch and left bundle branch. The left bundle branch includes left anterior fascicle and left posterior fascicle.
Under normal circumstances, notch T waves are more common in leads V2-V3 and are caused by asynchronous local ventricular repolarization.
The His bundle and the proximal bundle branches are mainly supplied by the 1st septal branch of the left anterior descending branch and the atrioventricular node artery of the right coronary artery.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
If the electrocardiogram during the onset of ventricular tachycardia cannot be recorded, it is possible to incorrectly analyze the QRS waveform based on the QRS waveform during the attack.
heart rhythm ekg note on paper Doctors use it to analyze heart disease treatments
In acute myocardial ischemia, the amplitude of T wave is increased first, and then the ST segment is elevated. When the end of QRS wave is deformed, there is a lack of collateral circulation.
Female doctor pointing to Heart rate monitor in operation room.Healthcare and Medical concept. Hospital and People theme.
Electromechanical separation is a kind of terminal ECG. The patient's ECG has electrical signals, the ECG wave is widened with morphological abnormalities, and the ventricle has no contraction.
Sometimes, left ventricular hypertrophy with tall T waves is easily misdiagnosed as hyperkalemia and hyperacute T waves, and ECG needs to be carefully identified in combination with clinic.
During left posterior fascicular block, the ECG showed right axis deviation. The QRS wave in leads I and aVL was rS wave, and the duration of QRS wave was less than 120 ms.
A 36 year old man survived CPR after sudden syncope. The electrocardiogram was suggestive of Brugada syndrome type 1. Implantation of ICD therapy.
Sometimes, because the QRS axis is in the upper left quadrant, the high-amplitude R wave of left ventricular hypertrophy occurs in the limb leads, and left chest leads is normal.
an abnormal electrocardiogram tracing (lateral wall ischemia) and a red heart-shaped object, heart disease concept, medical background, medical education
R wave greater than S wave is judged to be positive; R smaller than S is judged to be negative; R equal to S amplitude is judged to be equipotential.
Torsade de pointes refers to the pleomorphic ventricular tachycardia that occurs in the background of long QT interval, and the polarity of QRS wave twists around the equipotential line.
A patient with acute extensive anterior myocardial infarction developed ventricular tachycardia during hospitalization and quickly experienced cardiac arrest.
Heart beats Green cardiogram
Generally, when the sinus heart rate is below 60 beats per minute, it is called sinus bradycardia. This arrhythmia can be both physiological and often pathological.
Male, 60 years old, clinically diagnosed as acute extensive anterior wall myocardial infarction. The patient died of ventricular fibrillation after admission.
Note that the V3 lead of this ECG shows that the amplitude of R wave is greater than the amplitude of S wave, and there is counterclockwise rotation.
ECG with a heart attack in hospital ward
eart beats cardiogram
Male, 60 years old, clinically diagnosed as acute extensive anterior wall myocardial infarction. The patient died of ventricular fibrillation after admission.
Stethoscope and red toy heart lying on electrocardiogram on blue background closeup
Woman doctor showing heart model to students
watercolor heart health, and cardiogram. heart disease, heart rate. syringe treatment
Young team or group of doctors
watercolor heart health, and cardiogram. heart disease, heart rate.
Female, 51 years old, diagnosed with mitral stenosis. When this ECG was taken, the patient still maintained sinus rhythm.Note that the P wave duration was widened.
Doctors cardiologists looking at cardiogram on tablet and showing it with pen

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