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ECG
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In acute high lateral myocardial infarction, there is indicative ST segment elevation in leads I and aVL, and corresponding ST segment depression in leads II, III and aVF.
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The illustration shows the two patterns of ventricular tachycardia episodes.The green circle represents sinus rhythm. Picture A shows paroxysmal episodes of ventricular tachycardia, and picture B shows short bursts.
Bidirectional ventricular tachycardia is a kind of malignant arrhythmia. The polarity of QRS main wave alternates from beat to beat, and it is easy to degenerate into ventricular fibrillation.
A 36 year old man survived CPR after sudden syncope. The electrocardiogram was suggestive of Brugada syndrome type 1. Implantation of ICD therapy.
R wave greater than S wave is judged to be positive; R smaller than S is judged to be negative; R equal to S amplitude is judged to be equipotential.
Sometimes, because the QRS axis is in the upper left quadrant, the high-amplitude R wave of left ventricular hypertrophy occurs in the limb leads, and left chest leads is normal.
ECG monitoring of patient
During the onset of variant angina pectoris, ECG is divided into non fusion wave, partial fusion wave and complete fusion wave according to the fusion degree of QRS wave, ST segment and T wave.
Female, 51 years old, diagnosed with mitral stenosis. When this ECG was taken, the patient still maintained sinus rhythm.Note that the P wave duration was widened.
In case of acute anterior myocardial infarction, the characteristics of ST segment elevation in ECG can be used to deduce whether the culprit vessel system is the left main trunk or the proximal LAD.
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The left main coronary artery can be divided into the left anterior descending artery and the left circumflex artery, and sometimes the intermediate branch artery.
When the left free wall and septal accessory pathway are excited, preexcitation waves with different polarities are generated in leads and aVL.
Relative bradycardia refers to a pathophysiological phenomenon in which the patient's body temperature rises, but the pulse does not increase, which is common in some infectious diseases and jaundice.
It is best to measure the QRS wave duration in a 12 lead synchronous electrocardiogram, as some of the QRS wave start and end points are located on the isoelectric line.
When there is a the first degree interatrial block, the impulse from the right atrium is slowly transmitted to the left atrium, causing widening notched P wave.
The transverse vectorcardiogram generates a chest leads electrocardiogram, with the maximum ventricular excitation potential oriented towards the left posterior region.
When sinus arrest occurs, the electrocardiogram will show a long P-P interval, which is not multiples of the basal sinus cycle, including physiological and pathological reasons.
When the rhythm of the atria originates in the lower part of the atria, the whole atria are excited from inferior to superior, producing negative P waves in the inferior leads.
Male, 84 years old, admitted to hospital with chest pain for 1 day. These ECG rhythms are the Holter monitor records of the patients after admission, and they are third degree atrioventricular block.
The 4-phase membrane potential of sinoatrial node pacing exhibits spontaneous depolarization, while the 4-phase membrane potential of ventricular myocytes remains stable.
On the conventional 12-lead ECG, under normal circumstances, there are some inherent patterns of QRS waves in different leads, which are not exactly the same.
Under normal circumstances, in the chest lead electrocardiogram, the amplitude of the R wave gradually increases from lead V1 to lead V6.
Four anatomical malformations of tetralogy of Fallot: 1 aortic straddling; 2 ventricular septal defect; 3 right ventricular hypertrophy and 4 pulmonary artery stenosis.
In humans, Purkinje fibers are not distributed throughout the entire ventricular wall, but rather in the superficial myocardium beneath the endocardium and do not reach the epicardium.
When emphysema occurs, the diaphragm moves downwards, pulling the right atrium, causing an increase in the longitudinal longitude of the right atrium, and an increase in the amplitude of the sinus P wave.
QRS wave is a ECG wave generated by ventricular excitation, typically in a three-phase waveform, named qRs wave. The QRS waveform of each lead is different.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract forms a high amplitude R wave in the inferior leads and a QS wave or rS wave in the V1 lead.
When the left anterior wall and posterior wall accessory pathway are excited, preexcitation waves with different polarities are generated in the inferior wall leads of , and aVF.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
Male, 71 years old, was clinically diagnosed with upper gastrointestinal bleeding. During sleep at night, ECG monitoring showed sinus bradycardia, blood pressure 115 and 70mmHg.
Abnormal ECG refers to changes in depolarization waves and or repolarization waves, most of which are pathologic and few are physiological.
On the electrocardiogram, observing the morphology of QRS waves in lead V1 can distinguish whether ventricular pre excitation is located in the left ventricle or the right ventricle.
QRS wave is a ECG wave generated by ventricular excitation, typically in a three-phase waveform, named qRs wave. The QRS waveform of each lead is different.
When sinus bradycardia is obvious, the ventricle can be controlled by junctional escape and ventricular escape, and escape rhythm appears.
Surrounding the atrioventricular ring, except for the anterior septum of the left ventricle, there is no distribution of accessory pathways, and accessory pathways can exist in other parts.
On the electrocardiogram, the range of the myocardium is explored based on the leads, and some leads are grouped according to myocardial anatomy to form anatomically contiguous leads.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
Ventricular tachycardia originating from the right ventricular outflow tract can be sustained or short-burst, and is a benign idiopathic ventricular tachycardia.
When a 2:1 bundle branch block occurs, the refractory period of the bundle branch is longer than one basal cardiac cycle but shorter than two basal cardiac cycles.
When there is a left ventricular free wall bypass, the polarity of the ventricular preexcitation is positive in lead V1 and negative in lead aVL on the electrocardiogram.
Male, 13 years old, clinically diagnosed with secundum atrial septal defect. Note that the QRS wave in lead V1 of the electrocardiogram has a qR shape, indicating right ventricular hypertrophy.
Third degree atrioventricular block in young women may be congenital, with the block located on the atrioventricular node or above bifurcation of the His bundle.
Sometimes, there may be slight non-specific changes and normal variations in the electrocardiogram, which are often due to physiological reasons and have no clinical therapeutic significance.
Abnormal ECG refers to changes in depolarization waves and or repolarization waves, most of which are pathologic and few are physiological.
On the electrocardiogram, observing the morphology of QRS waves in lead V1 can distinguish whether ventricular pre excitation is located in the left ventricle or the right ventricle.
When the frontal QRS axis is at +57, the QRS amplitude of lead is the highest.The frontal electrical axis is almost perpendicular to aVL lead, so the algebraic sum of QRS amplitude is almost zero.
Narrow QRS wave and wide QRS wave

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