114 Bilder zum Thema "st segment" bei ClipDealer

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Segment of a colorful graffiti on a wall
Segment of a colorful graffiti on a wall
Segment of a colorful graffiti on a wall
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
orange slice
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
skyscrapers frankfurt
Illustration,Tafel mit Diagramme
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
Red suitcase with city inside
In case of acute anterior myocardial infarction, the characteristics of ST segment elevation in ECG can be used to deduce whether the culprit vessel system is the left main trunk or the proximal LAD.
During the onset of variant angina pectoris, ECG is divided into non fusion wave, partial fusion wave and complete fusion wave according to the fusion degree of QRS wave, ST segment and T wave.
Ventricular tachyarrhythmia includes many clinical types, some benign and some malignant. For malignant ventricular arrhythmias, patients are at risk of death.
The QT interval of ECG is from the beginning of QRS wave to the end of T wave, representing the total time of ventricular depolarization and repolarization.
A 14-year-old leukemic child had a sudden wide QRS tachycardia with a frequency of 167 bpm, and the rhythm was regular. After anti-arrhythmia treatment, the patient recovered to sinus rhythm.
R wave greater than S wave is judged to be positive; R smaller than S is judged to be negative; R equal to S amplitude is judged to be equipotential.
At present, there is a younger trend in patients with acute myocardial infarction, so it is important to check the ECG for acute chest pain in young people.
A 36 year old man survived CPR after sudden syncope. The electrocardiogram was suggestive of Brugada syndrome type 1. Implantation of ICD therapy.
Sometimes, left ventricular hypertrophy with tall T waves is easily misdiagnosed as hyperkalemia and hyperacute T waves, and ECG needs to be carefully identified in combination with clinic.
Due to the large mass of the left ventricle, the dominant excitation potentials of the left and right ventricles are oriented towards the left ventricle, i.e. towards the left, Inferior and posterior.
It is best to measure the QRS wave duration in a 12 lead synchronous electrocardiogram, as some of the QRS wave start and end points are located on the isoelectric line.
Relative bradycardia refers to a pathophysiological phenomenon in which the patient's body temperature rises, but the pulse does not increase, which is common in some infectious diseases and jaundice.
A normal electrocardiogram includes normal morphology, amplitude, and various measurements of duration and interval, normal electrical axis, and normal R wave progression.
Electric impulses can be conducted, but the conduction speed slows down, resulting in conduction delay and affecting the morphology of the P wave, PR interval, and QRS wave.
In the frontal plane lead system, when the initial vector of the QRS wave is directed downward, the positive initial QRS wave is recorded in leads II, III, and aVF.
Male, 84 years old, admitted to hospital with chest pain for 1 day. ECG showed acute inferior and posterior MI and possibly right MI. The patient died of ventricular fibrillation the next day.
When acute left main artery occlusion causes ST segment elevation myocardial infarction, it is often accompanied by extensive anterior and high lateral myocardial infarction.
A patient with acute inferior and anterior myocardial infarction(MI) caused by distal occlusion of the RCA and one day later, combined with occlusion of the LAD, caused high lateral and anterior MI.
When there is a the first degree interatrial block, the impulse from the right atrium is slowly transmitted to the left atrium, causing widening notched P wave.
The transverse vectorcardiogram generates a chest leads electrocardiogram, with the maximum ventricular excitation potential oriented towards the left posterior region.
When sinus arrest occurs, the electrocardiogram will show a long P-P interval, which is not multiples of the basal sinus cycle, including physiological and pathological reasons.
When the rhythm of the atria originates in the lower part of the atria, the whole atria are excited from inferior to superior, producing negative P waves in the inferior leads.
Male, 84 years old, admitted to hospital with chest pain for 1 day. These ECG rhythms are the Holter monitor records of the patients after admission, and they are third degree atrioventricular block.
The 4-phase membrane potential of sinoatrial node pacing exhibits spontaneous depolarization, while the 4-phase membrane potential of ventricular myocytes remains stable.
On the conventional 12-lead ECG, under normal circumstances, there are some inherent patterns of QRS waves in different leads, which are not exactly the same.
Under normal circumstances, in the chest lead electrocardiogram, the amplitude of the R wave gradually increases from lead V1 to lead V6.
In the spatial anatomy of the heart, the axis from the base of the heart to the apex of the heart is called the long axis, that is, the upper right side faces the lower left side.
The terminal excitation of the ventricle forms the final part of the S wave in lead V1, gradually returning to the isoelectric line, and forms a small S wave in lead V5.
Four anatomical malformations of tetralogy of Fallot: 1 aortic straddling; 2 ventricular septal defect; 3 right ventricular hypertrophy and 4 pulmonary artery stenosis.
When emphysema occurs, the diaphragm moves downwards, pulling the right atrium, causing an increase in the longitudinal longitude of the right atrium, and an increase in the amplitude of the sinus P wave.
QRS wave is a ECG wave generated by ventricular excitation, typically in a three-phase waveform, named qRs wave. The QRS waveform of each lead is different.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract forms a high amplitude R wave in the inferior leads and a QS wave or rS wave in the V1 lead.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
When the first degree interatrial block occurs, the conduction time from the right atrium to the left atrium is prolonged, the P wave widens, and bimodal P wave ECG changes appear.
A patient with acute inferior and anterior myocardial infarction(MI) caused by distal occlusion of the RCA and one day later, combined with occlusion of the LAD, caused high lateral and anterior MI.
In the aVR lead, the QRS wave can be in the form of QS, rS, Qr, rsr, etc., with the main wave being negative.
When the frontal QRS axis is at +83, the R amplitude of lead aVF is the highest.The frontal QRS axis is almost perpendicular to the axis of lead .
Clockwise rotation electrocardiogram refers to the transition of the rS waveform of the chest lead to the left chest lead, with the transition lead exceeding the V4 lead.
Some patients with severe sinus bradycardia have triggers that can disappear after treatment, while others are permanent and require treatment with ventricular pacemakers.
Septal q wave loss refers to the initial q wave loss of leads I, aVL, V5, and V6, which can be partially or completely lost.
Abnormal ECG refers to changes in depolarization waves and or repolarization waves, most of which are pathologic and few are physiological.
The initial excitation of the ventricle forms a small r wave in lead V1 and a small q wave in lead V6.
QRS wave is a ECG wave generated by ventricular excitation, typically in a three-phase waveform, named qRs wave. The QRS waveform of each lead is different.
The accompanying ST-T changing in the context of wide QRS complexes.The wide QRS complex changes the order of ventricular depolarization and secondary changes in the order of repolarization.
In ST segment elevation myocardial infarction, the ST-T of ECG will undergo a characteristic evolution process, and finally appear pathological Q wave, sometimes lasting for a lifetime.
In acute myocardial ischemia, the amplitude of T wave is increased first, and then the ST segment is elevated. When the end of QRS wave is deformed, there is a lack of collateral circulation.
On the electrocardiogram, the range of the myocardium is explored based on the leads, and some leads are grouped according to myocardial anatomy to form anatomically contiguous leads.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
Ventricular tachycardia originating from the right ventricular outflow tract can be sustained or short-burst, and is a benign idiopathic ventricular tachycardia.
When a 2:1 bundle branch block occurs, the refractory period of the bundle branch is longer than one basal cardiac cycle but shorter than two basal cardiac cycles.

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